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Schwann cells promote sensory neuron excitability during
Paradoxical hyperexcitability from NaV1.2 sodium channel loss in
Shmuel CHEN, MD PhD, Hebrew University of Jerusalem, Jerusalem, HUJI, Department of Cardiology
Subcellular expression and neuroprotective effects of SK channels
Enhancement of dendritic persistent Na+ currents by mGluR5 leads
Converging roles of ion channels, calcium, metabolic stress, and
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Effects of calcium-activated potassium channel modulators on
Role of Small Conductance Ca2+-Activated K+ Channels in
IJMS, Free Full-Text
PDF) Role of Small Conductance Ca2(+)-Activated K+ Channels in Controlling CA1 Pyramidal Cell Excitability